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. 2016 Feb 19;7(13):15868–15884. doi: 10.18632/oncotarget.7520

Figure 5. SGK1-dependent modulation of oxidative stress response.

Figure 5

A. Western blot analysis. SGK1 expression in p-HIV-EGFP ADF cells (left) and p-HIV-EGFP SGK1ADF cells (right). GAPDH was used as a loading control. B. The bar graphs represent the total number of p-HIV-EGFP ADF cells (left) or p-HIV-EGFP SGK1ADF cells (right), treated or untreated with either H2O2 (250 μM) for 2 h or SI113 (12.5μM) for 72 h. Results, obtained by means of Guava ViaCount assay, are expressed as the mean ± SE of six independent experiments for each cell line. Statistical significances are reported in the Tables under each graph.*P ≤ 0.05; **P ≤ 0.01; ***P ≤ 0.001. C. Western blot analysis. LC3B-I/LC3B-II conversion and GRP78 expression in p-HIV-EGFP ADF cells (left) and p-HIV-EGFP SGK1ADF cells (right), treated or untreated with H2O2 (250 μM) for 2h. GAPDH was used as a loading control.