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. 2016 Feb 24;7(13):16581–16592. doi: 10.18632/oncotarget.7667

Figure 4. Selinexor induced cell cycle arrest in GIST independent of KIT signaling pathway.

Figure 4

(A) Cell cycle analysis by propidium iodide staining in the GIST-T1 line and the GIST-T1/829 subclone. The cells were fixed following 24-hour exposure of each drug and analyzed by flow cytometry. (B) Protein expression analysis in the GIST-T1 line following 24-hour exposure of each drug. (C) Cell viability assay in the GIST-T1 line following the 72-hour exposure to the serial concentration of imatinib (IM) with or without 100 nM selinexor.