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a Examples of consecutive original traces of cardiac mCa1 in mitoplast from UCP3−/−: mCa1 in UCP3−/− control versus mCa1 in UCP3−/− + Ru360 versus mCa1 in UCP3−/− + ATP. b In cardiac mitoplasts from UCP3−/− Ru360 (10 µM, n = 5) significantly decreased total open probability (Po, total) of mCa1 (*P < 0.05). ATP (1 mM, n = 4) had no significant effect on mCa1 activity in UCP3−/−
