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. 2016 Jul 12;13:127. doi: 10.1186/s12985-016-0585-4

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Replication kinetics of the rX31 virus encoding PR8 NS1s (WT, G45R or R38AK41A (AA)) in wild type (WT) and type I IFN receptor null (IFNAR^null) Let1 cells. rX31 with G45R replicated to significantly higher titer that WT in both a WT and b IFNAR^null Let1 cells. The virus with RNA binding deficient NS1, AA, could not replicate well in both cell types. *** represents the statistically significant difference of mean titers compared to the WT at p < 0.001, as determined by ANOVA