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. 2016 Jun 30;2016:5938740. doi: 10.1155/2016/5938740

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Copyright © 2016 Ting-I Lee et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Cardiac fatty acid metabolic proteins in control, diabetes mellitus (DM), and MPT0E014-treated DM (DM + MPT0E014) rats. Representative immunoblots and average data of (a) ratio of phosphorylated 5′ adenosine monophosphate-activated protein kinase 2α (pAMPK2α) to total AMPK2α, (b) peroxisome proliferator-activated receptor- (PPAR-) γ coactivator- (PGC-) 1α, (c) phosphorylated acetyl coenzyme A carboxylase (pACC), (d) cluster of differentiation 36 (CD36), (e) diacylglycerol acyltransferase 1 (DGAT1), and (f) DGAT2 from control (n = 4), DM (n = 4), and DM + MPT0E014 (n = 4) rats. Densitometry was normalized to glyceraldehyde 3-phosphate dehydrogenase (GAPDH) as an internal control.