Skip to main content
. Author manuscript; available in PMC: 2017 Aug 1.
Published in final edited form as: Crit Care Med. 2016 Aug;44(8):e651–e663. doi: 10.1097/CCM.0000000000001637

Figure 1.

Figure 1

SRT1720 restores cellular energy status after hepatic ischemia-reperfusion (I/R). Ischemic liver tissues of the vehicle- and SRT1720-treated mice were collected at 24 h after reperfusion. Relative expression of the mitochondrial proteins (A) succinate dehydrogenase subunit B (SDHB) and (B) cytochrome oxidase IV (COXIV) as well as the autophagy markers (C) microtubule associate protein 1 light chain 3 (LC3) and (D) sequesterome 1 (SQSTM1) were measured in the liver by Western blot. (E) Adenosine triphosphate (ATP) levels in the liver lysates were measured. Data are presented as means ± SE (n = 6-8/group). *P < 0.05 versus control; #P < 0.05 versus vehicle.