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. 2016 Jul 18;4:22. doi: 10.1186/s40635-016-0096-z

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 7 — Aspect of peritoneal cavity of the sham-operated mice (a, c, e) and the 20 % ligated mice (b, d, f) 24 h after surgery. a The cecum is identified by a white star. b The 20 % ligated animal with cecal abscess (). When observing liver macroscopic morphology, we found a patchy appearance corresponding to pale ischemic areas (d) in contrast to the sham liver (c). f These areas were centrilobular necrosis of hepatocytes (asterisk) (e and f hematoxylin-eosin coloration ×50)

Inline graphic — Aspect of peritoneal cavity of the sham-operated mice (a, c, e) and the 20 % ligated mice (b, d, f) 24 h after surgery. a The cecum is identified by a white star. b The 20 % ligated animal with cecal abscess (). When observing liver macroscopic morphology, we found a patchy appearance corresponding to pale ischemic areas (d) in contrast to the sham liver (c). f These areas were centrilobular necrosis of hepatocytes (asterisk) (e and f hematoxylin-eosin coloration ×50)