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. 2015 Sep 17;71(1):115–118. doi: 10.1111/all.12711

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© 2015 The Authors. Allergy Published by John Wiley & Sons Ltd.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Figures 1 and 2 — Time courses (x‐axes) of cyclosporin A treatment, skin symptoms, and IgE antibody reactivities to autoallergens and exogenous allergens in two AD patients. (A) Cyclosporin A dose (y‐axes: mg/kg bodyweight/day), (B) SCORAD documentation of skin manifestations (y‐axis: SCORAD indices), and (C) IgE autoreactivity to nitrocellulose‐blotted human epithelial cell extracts are displayed for different time points of blood sampling. Molecular weights (kDa) are shown at the left margin and (D) time course of specific serum IgE levels (y‐axes: kUA/L) to exogenous allergens (mite allergens, recombinant major birch pollen allergen, rBet v 1, and two recombinant major timothy grass pollen allergens, rPhl p 1 and rPhl p 5).