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. 2016 Jul 19;16:154. doi: 10.1186/s12866-016-0777-5

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — CXCL8 responses of human nasopharyngeal epithelial cells to pneumococcal strain D39 (serotype 2) and its mutants lacking pneumolysin (D39Δply) or capsule (D39Δcps) or both (D39ΔplyΔcps) in the absence and presence (1 mg or 100 μg) of liposomes. Liposomes reduced CXCL8 levels following exposure to the wildtype strain D39 strain but also reduced the CXCL8 response to its mutants lacking pneumolysin. * indicates significant difference. Error bars indicate standard deviation