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. 2016 Jul 20;10:200. doi: 10.1186/s13256-016-0989-1

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Cerebral scans (axial slices, I–IV) obtained by 2-deoxy-2-[fluorine-18]fluoro-D-glucose integrated with computed tomography-positron emission tomography. Cerebral 2-deoxy-2-[fluorine-18]fluoro-D-glucose integrated with computed tomography-positron emission tomography was performed 23 months before the patient’s death. a Four representative axial slices (AI–AIV) show decreased fluorodeoxyglucose uptake in the frontal and temporal lobes and normal uptake in the posterior cingulum and occipital cortex. Note that the hypometabolism in the affected regions is greater in the right hemisphere. b Cerebral 2-deoxy-2-[fluorine-18]fluoro-D-glucose integrated with computed tomography-positron emission tomography performed 3 months before the patient’s death shows the general reduction in radiotracer uptake in the same axial levels (BI–BIV)