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. 2016 May 4;12:1744806916646381. doi: 10.1177/1744806916646381

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© The Author(s) 2016

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 3.0 License (http://www.creativecommons.org/licenses/by-nc/3.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access page (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 1. — Enhanced neuropathic pain and decreased expression of GRK6 in CCI rats. (a) CCI induces a significant decrease in PWT from 3 days to 21 days after surgery in the ipsilateral side. *p < .05, compared with Sham group. (b) The development of heat hyperalgesia from 3 days to 21 days was induced by CCI in the ipsilateral side. *p < .05, compared with Sham group. (c) Expression of GRK6 protein in DRGs was decreased from 3 days to 21 days after surgery in the ipsilateral side with the lowest level seven days after CCI. *p < .05, compared with Sham group. (d) GRK6 mainly expressed in neurons co-localized with β-tubulin, IB4, and CGRP in DRGs, but not in glial cells stained with GFAP. Bar = 75 µm.