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. 2016 May 4;12:1744806916646381. doi: 10.1177/1744806916646381

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© The Author(s) 2016

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 3.0 License (http://www.creativecommons.org/licenses/by-nc/3.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access page (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 2. — Suppression of neuropathic pain by GRK6 overexpression. (a) GFP image showed the successfully infection of lentiviral in DRG by sciatic nerve injection. (b) Western blot analysis presented the enhanced expression of GRK6 in DRG from CCI rats after GRK6 lentiviral injection. (c) GRK6 lentiviral injection, rather than NC lentiviral, markedly increased PWT on the CCI rats. *p < .05, compared with CCI group. (d) GRK6 lentiviral injection, rather than NC lentiviral, markedly increased PWL on the CCI rats. *p < .05, compared with CCI group. LV-GFP: lentiviral carrying the green fluorescent protein; CCI: chronic constriction injury; NC-LV: Negative control lentivirus; PWL: paw withdraw latency; PWT: paw mechanical withdrawal thresholds.