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. 2016 Jul 20;8(7):380. doi: 10.3390/nu8070380

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© 2016 by the authors; licensee MDPI, Basel, Switzerland.

This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC-BY) license (http://creativecommons.org/licenses/by/4.0/).

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EGCG attenuated tobacco smoke-induced alterations of epithelial-mesenchymal transition markers in the stomach of mice. (A) qRT-PCR analyses of E-cadherin, ZO-1, CK5, Snail-1, Vimentin, and N-cadherin mRNAs; (B) Western blotting analyses of E-cadherin, ZO-1, Snail-1, Vimentin, and N-cadherin proteins; (C) Densitometric analyses of Western blotting. Data are expressed as mean ± SD. ** p < 0.01, compared with FA control; ^# p < 0.05, compared with TS; ^## p < 0.01, compared with TS. FA = filtered air; TS = tobacco smoke.

EGCG attenuated tobacco smoke-induced alterations of epithelial-mesenchymal transition markers in the stomach of mice. (A) qRT-PCR analyses of E-cadherin, ZO-1, CK5, Snail-1, Vimentin, and N-cadherin mRNAs; (B) Western blotting analyses of E-cadherin, ZO-1, Snail-1, Vimentin, and N-cadherin proteins; (C) Densitometric analyses of Western blotting. Data are expressed as mean ± SD. ** p < 0.01, compared with FA control; ^# p < 0.05, compared with TS; ^## p < 0.01, compared with TS. FA = filtered air; TS = tobacco smoke.