Table 2.

Different results on various suPAR studies in FSGS and other glomerulopathies.

Negative results	References	Positive results	References
Serum suPAR levels did not correlate to eGFR	[9, 13]	Serum suPAR levels were inversely correlated with eGFR	[10, 14–24]
Serum suPAR levels did not correlate to CRP levels	[10, 13, 17]	Serum suPAR levels were positively correlated with CRP levels	[16, 18]
Serum suPAR levels are not influenced by immunosuppression	[17, 18]	Serum suPAR levels are influenced by immunosuppression	[10, 93]
Serum suPAR levels are not influenced by subtypes of FSGS	[11, 13, 89]	Serum suPAR levels are influenced by subtypes of FSGS	[12]
		Urine suPAR levels are influenced by subtypes of FSGS	[67]
Serum suPAR levels are not different between primary and secondary FSGS	[12]	Serum suPAR levels are higher in primary FSGS than in secondary FSGS	[89]
		Urine suPAR levels are higher in primary FSGS than in secondary FSGS	[67]
Serum suPAR levels did not predict response to steroids	[14, 18]	High serum suPAR levels predicted better response to steroids	[13]
High serum suPAR levels were not associated with acute tubulointerstitial lesions	[13]	High serum suPAR levels were associated with >50% interstitial fibrosis	[12]

suPAR, soluble urokinase-type plasminogen activator receptor; FSGS, focal segmental glomerulosclerosis; CRP, C-reactive protein; eGFR, estimated glomerular filtration rate.