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. 2016 Jul 11;113(30):E4377–E4386. doi: 10.1073/pnas.1600015113

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Fig. S5. — smn-1(RNAi) knockdown causes endocytic defects in coelomocytes. (A) Representative images of animals injected with coelomocyte-specific smn-1 RNAi (unc-122p::smn-1RNAi) or empty control (unc-122p::emptyRNAi). (B) Pie charts illustrate the percentage of animals with indicated level of GFP accumulation in the body cavity. GFP is secreted from muscle cells; coelomocyte-specific knockdown of smn-1 resulted in a significant accumulation of GFP in the body cavity and increased GFP intensity in coelomocytes, consistent with a cell-autonomous endocytic defect. No gross morphology changes and no large vacuoles were observed. Line indicates 200 microns.