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. 2016 Aug 2;16:567. doi: 10.1186/s12885-016-2632-3

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Ectopic expression of p44/PRMT5-target genes partially restores growth defect of p44-silencing cells. a Western blot analysis of whole-cell lysates of A549 cells expressing NT (lane 1) or p44 shRNA (lanes 2-4) infected with lentivirus expressing ErbB3 (lanes 3 and 4). b Expression of ErbB3 increased growth of p44-silenced cells. c Western blot analysis of whole-cell lysates derived from A549 cells expressing GLIPR1, FGFR3 or the combination. d Growth of A549 cells expressing GLIPR1 or GLIPR1 plus FGFR3