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. 2016 Aug 3;7:1192. doi: 10.3389/fmicb.2016.01192

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Copyright © 2016 Xiong, Wang, Tian and Tian.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Loss of VdMcm1 causes defects in microsclerotia formation. (A) Microsclerotia formation on BM plates with 10⁵ conidia spreading over the cellulose membrane after 6 days of growth. The enlarged view shows the unmelanized and swollen hyphae of ΔVdMcm1-5. The scale bars represent 100 μm. (B) The histogram represents the number of microsclerotia formed by the wild-type strain, ΔVdMcm1-5, and complemented strain on the cellulose membrane (φ = 80 mm) after 6 days of growth. (C) Microsclerotia formation on BM plates after 8 days of incubation. The enlarged view showed the microsclerotia of ΔVdMcm1-5. The scale bars represent 100 μm. (D) The histogram represented the number of microsclerotia formed by wild-type strain, ΔVdMcm1-5, and complemented strain on the cellulose membrane (φ = 80 mm) after 8 days of incubation. The error bars represent standard deviations. The experiments were performed in triplicate.