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. 2016 Jul 21;7(7):e2300. doi: 10.1038/cddis.2016.213

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Copyright © 2016 The Author(s)

Cell Death and Disease is an open-access journal published by Nature Publishing Group. This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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Loss of Msi2-induced apoptosis in pre-osteoclasts. (a) Cell cycle progression in BMM was measured by flow cytometry using 7′AAD. The average percentage of cell number in each phase of cell cycle is shown in the graphs. (b) The control and Msi2 knockdown BMM or pOC were serum/cytokine starved for 3 h. The apoptosis rate was measured by a cell death ELISA. Difference was analyzed by one-way ANOVA (versus Luc-sh) (**P<0.01) (c) Luc-sh and Msi2-sh1 and sh2 BMM or pOC were either untreated or serum/cytokine starved for 3 h. The cleaved caspase 3 and PARP, two markers of apoptosis, was detected by western blots. Actin served as a loading control