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Pressure volume loop measurements. AAV-LUC controls (n=9) showed a significantly impaired systolic left-ventricular function, noticeably reduced end-systolic pressure (ESP, a), decreased stroke work (SW, b) and lower pressure development rates (dP/dtmax, c) when compared with wild-type mice (n=7) and AAV-DES-treated animals (n=8). *P<0.05.
