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. 2004 Aug;42(8):3635–3643. doi: 10.1128/JCM.42.8.3635-3643.2004

TABLE 2.

PCR ribotypes of C. difficile isolates from 330 episodes of CDAD in Örebro County during 12 months

PCR ribotypea Serotype matchb No. of episodes (% of isolates [n = 330])
Primary
Recurrencesc All
Hospital-associated
Community-acquired All
Hospital onset Community onset All
Major types
    SE7 4 0 4 3 7 4 11
    SE12 A2 9 1 10 5 15 9 24
    SE16 A 5 3 8 3 11 1 12
    SE17 C 33 (21) 9 (29) 42 (22)f 3 (6) 45 (19) 20 (22) 65 (20)
    SE20 G 9 3 12 3 15 8 23
    SE21 H 16 3 19 6 25 6 31
    SE21b A8 18 1 19 3 22 3 25
    SE25 7 1 8 2 10 6 16
    SE30 5 0 5 2 7 1 8
    All (n = 9) 106 (67) 21 (68) 127 (67) 30 (59) 157 (65) 58 (65) 215 (65)
Minor types (n = 23)d 44 7 51 14 65 28 93
Unique types (n = 21)e 9 (6) 3 (9) 12 (6) 7 (14) 19 (8) 3 (3) 22 (6)
All types (n = 53) 159 (100) 31 (100) 190 (100) 51 (100) 241 (100) 89 (100) 330 (100)g
a

Current Swedish designations. Major types were each found in 7 to 65 episodes.

b

Identity between the PCR ribotype of a C. difficile serotype reference strain and that of a major SE type. The PCR ribotype of other major SE types was distinctly different from that of each of the 22 serotype reference strains tested (see Materials and Methods). None of the isolates belonged to the PCR ribotype known to be toxin A-B+.

c

Apparent relapse or apparent reinfection (see Table 5).

d

Each type found in two to six episodes.

e

Each type found in only one episode.

f

Value statistically different from the value in the next column at a P value of 0.008.

g

Isolates for typing were not available in 42 episodes.