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. Author manuscript; available in PMC: 2016 Aug 8.
Published in final edited form as: Nat Neurosci. 2012 Jun 17;15(7):979–987. doi: 10.1038/nn.3135

Figure 2. mbc, dced-12, and rac1 mutants exhibit dominant genetic interactions in ORN axon clearance assays.

Figure 2

(a) OR85e-expressing ORN axons were labeled in control (w;OR85e-mCD8::GFP/+) animals and in mbcC1, dced-12KO, or rac1J11 heterozygous mutant backgrounds, maxillary palps were ablated, or left uninjured, and clearance of axons was assayed after 5 days (anti-GFP, green).

(b) Normalized quantification of data to uninjured cohorts from (a) Error bars represent s.e.m.; n>10; *** p<0.0001.