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. 2016 Aug 10;68(3):726–735. doi: 10.1161/HYPERTENSIONAHA.116.07911

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© 2016 The Authors.

Hypertension is published on behalf of the American Heart Association, Inc., by Wolters Kluwer. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDervis License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 4. — Genomic and nongenomic pathways of vascular mineralocorticoid receptor (MR) activity after isoproterenol treatment. Nuclear translocation of MR (A), mRNA expression of osteopontin (B), and protein expression of γ-epithelial sodium channel (γENaC; C), Src (D), and ERK1/2 (E) phosphorylation in thoracic aorta of control (CT) and isoproterenol (ISO) groups without or with spironolactone (SPI) treatment. Data represent mean±SEM; number of animals used in each group is indicated into the bars. Two-way ANOVA: P<0.05 *vs CT; #vs ISO.