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. 2015 Oct 19;1:15044. doi: 10.1038/cddiscovery.2015.44

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Copyright © 2015 Cell Death Differentiation Association

This work is licensed under a Creative Commons Attribution 4.0 International License. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license, users will need to obtain permission from the license holder to reproduce the material. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/

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The H1047R mutation in the p110α kinase domain of PI3K increases cell migration. The migratory capability of HCT116 WT and MUT cells was measured by wound-healing assay. HCT116 MUT cells revealed the faster wound closure compared with WT cells at 24 h.