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. 2016 Aug 3;53(3):753–771. doi: 10.3233/JAD-160165

Fig.4.

Fig.4

Long-term EMF treatment of aged AD (Tg) mice dramatically increased soluble Aβ1–40 levels in mitochondria preparations from both cortex and hippocampus. These 5–10x increases in mitochondrial Aβ are consistent with an EMF-induced disaggregation of toxic Aβ oligomers associated with intraneuronal mitochondria.