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. Author manuscript; available in PMC: 2016 Aug 15.
Published in final edited form as: Physiol Res. 2015 Jun 5;64(6):857–873. doi: 10.33549/physiolres.932977

Figure 9.

Figure 9

Left ventricle peak pressure (a), left ventricle end-diastolic volume (b), maximum rates of pressure rise [+(dP/dt)max] (c), maximum rates of pressure fall [−(dP/dt)max] (d) in HanSD rats treated with soluble epoxide hydrolase inhibitor (sEHi) (HanSD + sEHi), in HanSD rats treated with angiotensin-converting enzyme inhibitor (ACEi) (HanSD + ACEi), in untreated HanSD rats with aorto-caval fistula (ACF HanSD + water), in ACF HanSD rats treated with sEHi (ACF HanSD + sEHi) and in ACF HanSD rats treated with ACEi (ACF HanSD + ACEi). * P<0.05 compared with HanSD + water. # P<0.05 compared with * marked values.