Skip to main content
. Author manuscript; available in PMC: 2017 Jan 1.
Published in final edited form as: Glia. 2015 Sep 4;64(1):105–121. doi: 10.1002/glia.22906

Fig. 6. The number of Dcx+ cells in the corpus callosum was greater in Gal-3−/− mice after TMEV infection.

Fig. 6

(A) Forebrain schematic showing location of analysis for SVZ neuroblast emigration into the corpus callosum (cc) and striatum (str). (B–C) Dcx immunofluorescence in the corpus callosum and SVZ of C57BL/6 WT and knockout mice. Arrowheads point to Dcx+ cells in the corpus callosum. (D–E) fewer Dcx+ cells were found in the striatum of C57BL/6 Gal-3−/− mice after TMEV. Arrowhead points to Dcx+ cell in the striatum. Scale bar: F=20 μm. (F) The number of Dcx+ cells increased in the corpus callosum in C57BL/6 Gal-3−/− mice compared to WT. The number of Dcx+ cells decreased in the striatum in C57BL/6 Gal-3−/− mice compared to WT. (G) The number of Dcx+ cells increased in the corpus callosum in SJL/J Gal-3−/− mice compared to WT SJL/J. The number of Dcx+ cells in the striatum in SJL/J Gal-3−/− mice was not significantly different compared to WT SJL/J’s.*P<0.05 Gal-3−/− compared to WT, ** P<0.01, ***P<0.001, $ P<0.05 TMEV treated compared to shams, $$ P<0.01.