Fig. 1.

Initial AH studies focused on resting connectivity in auditory and language regions (upper figure), primarily identifying atypical connectivity of left posterior STG (a), PAC (b), and the TPJ area (c). Findings of atypical resting connectivity between left IFG (d) and STG are inconsistent although both areas are often implicated during AH. More recent findings implicate atypical interaction of the DMN, SN, and CEN in those prone to AH (lower figures). The combination of atypical DMN interaction with SN (1) and CEN (2) and altered resting connectivity in sensory areas could prompt the collapse of internally focused states into activation of auditory cortex (3), which is then reverberated along a frontotemporal loop. The IFG, STG, and surrounding areas are often implicated in symptom-capture studies.⁴⁸ Note: ACC, anterior cingulate cortex; AH, auditory hallucination; CEN, central executive network; DMN, default mode network; IFG, inferior frontal gyrus; PAC, primary auditory cortex; SN, salience network; STG, superior temporal gyrus; TPJ, temporoparietal junction.