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. 2016 Aug 18;11:60. doi: 10.1186/s13024-016-0127-y

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — Gene expression analysis of control and ATM-deficient human and mouse brains revealed that ATM deficiency had a huge impact on genes known to take part in brain glutamine metabolism. Presented are a list of genes whose expressions were found to be significantly reduced or increased in ATM-deficient human or mouse brains (>1.5 fold change when compared to control brains, p < 0.05). Upregulated genes are indicated by an black upward arrow next to the gene name; downregulated genes are shown by a downward arrow. Genes whose direction of change was significant in both mouse and human data are shown with underlines