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. 2016 Aug 22;13(1):28. doi: 10.1186/s12950-016-0136-1

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Hepatocyte proliferation is transiently suppressed in CCR2-deficient mice. a Percentage (mean +/− SEM) of BrdU⁺ hepatocyte nuclei from wild type or CCR2 knockout mice at the indicated times after PH. Mice were pulsed with BrdU 2 h prior to euthanasia at the indicated time points (n = 4). b Liver-body weight ratios (mean +/− SEM) of wild type and CCR2 knockout mice at the indicated times after PH. * p <0.05 when compared to wild type mice at same time point based on two-way ANOVA followed by Bonferroni’s post-hoc test