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. 2016 Jun 30;12(3):1279–1286. doi: 10.3892/etm.2016.3490

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — The expression levels of IL-1β and β-actin in the intestine of the mice from each group. (A) Blank control; (B) normal saline; (C) propylene glycol; (D) 0.2 mg/kg triptolide; (E) 0.4 mg/kg triptolide; (F) 0.6 mg/kg triptolide; and (G) dexamethasone treatment groups. The ratio of IL-1β protein to β-actin as determined by densitometry is shown. The data are presented as the mean ± standard deviation. *P<0.01, vs. the expression levels in the propylene glycol and normal saline treatment groups. IL, interleukin.