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. 2016 Jul 15;12(3):1896–1900. doi: 10.3892/ol.2016.4870

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — JNK inhibitor, SP600125, reverses the viability and apoptosis of suspended HUVECs induced by DHA. (A) Representative immunoblots of p-JNK and JNK in suspended HUVECs treated with DHA and SP600125. (B) Percentage of viable cells from suspended HUVECs treated with DHA and SP600125; n=4. (C) Representative images of flow cytometry analyses of Annexin V/PI-staining in suspended HUVECs treated with DHA and SP600125. DHA, dihydroartemisinin; JNK, c-Jun N-terminal kinase; p-JNK, phosphorylated-JNK; GAPDH, glyceraldehyde 3-phosphate dehydrogenase; HUVEC, human umbilical vein endothelial cell; n.s., non-significant; PI, propidium iodide; FITC, fluorescein isothiocyanate.