Table 2.
Comparison of the inhibiting efficiency of bacteriocin-producing strain C. divergens 41 (div−) and the non-bacteriocin-producing strain V41C9 (div−) on L. monocytogenes Scott A plaque-forming on HT29 and mucus-secreting HT29 MTX.
| C. divergens | L. monocytogenes (cfu ml−1) | HT29 | HT29-MTX |
|---|---|---|---|
| Mean ± SD (%) | Mean ± SD (%) | ||
| V41 (div+) | Scott A (107) | 100.0 ± 0.0 | 100.0 ± 0.0 |
| V41 (div+) | Scott A (108) | 86.5 ± 6.3 | 89.1 ± 1.7 |
| V41C9 (div−) | Scott A (107) | 91.5 ± 6.6* | 98.7 ± 2.7 |
| V41C9 (div−) | Scott A (108) | 74.9 ± 4.4* | 81.7 ± 3.2* |
Carnobacteria were inoculated at 109 cfu ml-1. HT29 and HT29 MTX epithelial cells were precoated with Carnobacteria 1 h before the inoculation of L. monocytogenes at 107 or 108cfu ml-1. Results were normalized to the plaques obtained with L. monocytogenes Scott A alone (log 3.27 ± 0.12, n = 4 on HT29 and log 2.80 ± 0.08, n = 4 on HT29 MTX). The means and standard deviations (SD) were calculated from at least three independent experiments. Asterisks indicate significant differences (P < 0.05) between div+ and div−.