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. Author manuscript; available in PMC: 2017 Sep 1.
Published in final edited form as: J Trauma Acute Care Surg. 2016 Sep;81(3):512–519. doi: 10.1097/TA.0000000000001137

Figure 3.

Figure 3

Tubastatin-A induces acetylation of both H3K9 and α-tubulin in Caco-2 cells after anoxia. (A) Tubastatin A induces aceylation of H3K9 after anoxia (*P=0.002 compared to Sham, *P=0.008 compared to control), no differences were found between sham and control group. (B) Tubastatin A induces acetylation of α-tubulin (*P=0.004 compared to sham, *P=0.004 compared to control). No differences were found between sham and control. Protein bands quantified by densitometry were expressed as mean values ± SD. Ac-H3K9: acetylated histone 3 at lysine 9; Ac-α-Tubulin: acetylated α-Tubulin; Sham: no anoxia and no treatment; Control: anoxia, no treatment; Tub-A: Tubastatin-A treatment.