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. 2016 Sep;148(3):195–199. doi: 10.1085/jgp.201611669

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© 2016 Wingo and Stockand

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Figure 1. — The metabolic alkalosis of Bartter’s syndrome can arise from an increase in acid secretion from α intercalated cells and a decrease in base secretion from β intercalated cells. Depiction of the renal nephron showing the cells and transporters involved in the renal salt wasting and alkalosis of Bartter’s syndrome. CD, collecting duct; CNT, connecting tubule; DCT, distal convoluted tubule; NCC, sodium chloride cotransporter; ROMK, renal outer medullary potassium channel; TAL, thick ascending limb.