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LCB2 and SUR2 response to elevated Pdr pathway activity and rox1Δ. LCB2-lacZ and SUR2-lacZ fusions carried on low-copy-number plasmids were introduced into isogenic wild-type or rox1Δ strains along with an empty vector (pRS315) or the same plasmid carrying the hyperactive PDR3-11 allele. Transformants were grown and assayed for β-galactosidase activity as described in the text.
