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. 2016 Sep 5;10:210. doi: 10.3389/fncel.2016.00210

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Copyright © 2016 Zwarts, Goossens, Clements, Kang and Callaerts.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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PlexA and PlexB are involved in MB development. (A) UAS-RNAi-PlexA;UAS-mCD8-Gfp;OK107-Gal4, (A) anti-FasII staining (magenta), (A′) GFP (green), (A′′) overlay. The α lobes show outgrowth defects (arrows). (B) PlexB^KG0087, anti-FasII staining (magenta). One α lobe is shorter and shows misorientation (arrow). (C) UAS-RNAi-PlexB;;OK107-Gal4, anti-FasII staining. Knock-down of PlexB in the MB causes no neuroanatomical MB defects.