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. 2016 Sep 5;16:67. doi: 10.1186/s12935-016-0342-1

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — SMAD3 protein levels are down-regulated by miR-23a. a SMAD3 protein expression was decreased by treatment with the miR-23a agomir but increased by treatment with a miR-23a antagomir in HEC-1-A cells (top data from the gels; bottom normalization to GAPDH) (n = 6 per group) (*P < 0.05). b SMAD5 protein expression was decreased by treatment with the miR-23a agomir but increased by treatment with a miR-23a antagomir in HEC-1-A cells (top data from the gels; bottom normalization to GAPDH). SMAD5 protein level was not significantly influenced by the overexpression or inhibition of miR-23a (n = 12 per group)