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. 2016 Jul 25;291(37):19364–19373. doi: 10.1074/jbc.M116.732347

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© 2016 by The American Society for Biochemistry and Molecular Biology, Inc.

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FIGURE 6. — PKC mediates the paclitaxel-induced activation of presynaptic NMDARs at primary afferent terminals. A, original current traces show the lack of effect of 50 μm AP5 on the amplitude of monosynaptic EPSCs of a lamina II neuron from a spinal cord slice pretreated with 10 μm chelerythrine in a vehicle-treated rat and a paclitaxel-treated rat. B, group data show the effect of AP5 on the amplitude of evoked EPSCs in spinal cord slices pretreated with chelerythrine in vehicle-treated (n = 11 neurons, 4 rats) and paclitaxel-treated rats (n = 10 neurons, 3 rats).