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. 2016 Sep 8;9(1):84. doi: 10.1186/s13041-016-0264-9

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Increased pThr514 CRMP2 correlates with β-III-tubulin loss in in LBD parietal cortex. a Bar graph of β-III-tubulin immunoreactivity (mean ± SEM in arbitrary units) and representative immunoblots, with GAPDH as loading control. Scatter plots of pThr514 CRMP2 with β-III-tubulin immunoreactivities in total homogenate fractions of b LBD (DLB + PDD), c DLB and d PDD parietal cortex, with insets indicating rho and p values. Where a correlation is significant, a best-fit regression line (solid line) is added together with its 95 % prediction intervals (dotted lines). Available N for control (C) = 19; PDD (P) = 19 and DLB (D) = 20. ^§§ p < 0.01, significant difference for multiple pair-wise comparisons (one-way ANOVA with Bonferroni post-hoc tests). *p < 0.05, significant Spearman correlation