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. 2016 Sep 13;18(1):204. doi: 10.1186/s13075-016-1098-7

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Increased expression of adhesion molecule CD54 (ICAM-1), chemokine receptor CCR5 and NKG2C on CD56^dim natural killer (NK) cells in active (non-remission) granulomatosis with polyangiitis (GPA). Experimental setting, patients and analysis as described in Fig. 3, using multicolor panel III. a Left mean fluorescent intensity (MFI) of CD54 on CD56^dim NK cells; Kruskal-Wallis test, p = 0.0108; middle percentage of CCR5-positive CD56^dim NK cells (Kruskal-Wallis test, p = 0.0204); right percentage of CXCR3-positive CD56^dim NK cells (Kruskal-Wallis test not significant). b Left percentage of NKG2C on CD56^dim NK cells (Kruskal-Wallis test, p = 0.0227); right percentage of NKG2C on CD56^bright NK cells (Kruskall-Wallis test not significant). a, b Dunn's post hoc test was significant where indicated in the graphs. HC healthy controls