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. 2016 Jun 2;44(16):7722–7741. doi: 10.1093/nar/gkw506

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© The Author(s) 2016. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 2. — High-copy HOP1 specifically suppresses the checkpoint defect of zip1 pch2, zip1 sir2 and zip1 dot1. (A) Dityrosine fluorescence assay of the indicated strains transformed with empty vector (YEp352) of high-copy HOP1 (R1692) after 3 days on sporulation plates. (B) Microscopic quantification of the sporulation efficiency of the strains analyzed in (A). Three independent counts were performed. Means and standard deviations are shown. (ns), no significant difference; (***), P < 0.001. Strains are: BR2495 (wild type), MY63 (zip1), DP174 (zip1 dot1), DP223 (zip1 pch2), DP267 (zip1 sir2), S4295 (zip1 rad24), S4278 (zip1 ddc1) and S4286 (zip1 rad17).