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. 2016 Sep 21;8:220. doi: 10.3389/fnagi.2016.00220

Figure 2.

Figure 2

CCM lesions diagnosed through CT, GE-and SWI-MRI. (A) CT of the 57-year-old proband, II-1. A plain CT scan (brain window images) showed multiple calcification spots scattered on the tegmentum of the pons, temporal lobes and periventricular area. (B) Axial T1-weighted gradient-echo (GE) MR imaging of the proband, II-1. Axial T1-weighted GE images demonstrated hyper-intensity of hemorrhage lesions. (C) Axial T2-weighted gradient-echo (GE) MR imaging of the proband, II-1. T2-weighted GE images showed a “popcorn” appearance surrounded by a dark rim of hemosiderin. (D) SWI-MR imaging of the proband, II-1. SWI revealed thickly dotted CCMs distributed throughout the cerebral cortex in the brain of the proband. There are dozens of lesions on the cerebrum, cerebellum, thalamus, and brain stem. The diameter of the lesions ranges from a few millimeters to several centimeters. (E) SWI-MR imaging of the proband's elder brother, II-2. II-2 showed a similar phenomenon, with multiple CCM lesions observed upon SWI-MRI of the brain. (F) SWI-MR imaging of the proband's younger sister, II-3. II-3 did not show any CCM lesions upon SWI-MRI of the brain.