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. 2016 Sep 22;9:93. doi: 10.1186/s13045-016-0327-5

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Alantolactone induced the apoptosis and differentiation of leukemia stem-like cell line KG1a and primary AML cells. a The expression of CD34⁺CD38^– on the surface of KG1a cells by flow cytometry. The χ axis represented CD34-APC, and the Y axis represented CD38-AF488. b Alantolactone induced the differentiation of leukemia stem cells after 3 days treatment. c Alantolactone induced apoptosis of primary AML cells with a dose-dependent manner after the treatment of alantolactone for 18 h. Each point represents an AML specimen. d Alantolactone induced apoptosis with a dose-dependent manner in KG1a cells with the treatment of alantolactone for 24 h. Analysis results represented mean ± SD based on three independent experiments, **P < 0.01, ***P < 0.0001