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. 2016 Aug 29;12(4):2515–2520. doi: 10.3892/etm.2016.3625

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Measurement of uterine artery pulsatility index (PI) and serum markers. (A) Second-trimester uterine artery PI in 39 pregnancies which developed preeclampsia and in 100 normal controls. (B) Second-trimester inhibin A levels in 39 pregnancies which went on to develop preeclampsia and in 100 normal controls. (C) Second-trimester activin A levels in 39 pregnancies which went on to develop preeclampsia and in 100 normal controls. (D) Placental growth factor (PlGF) levels in 39 pregnancies which went on to develop preeclampsia and in 100 normal controls (***P<0.001).