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. 2016 Aug 22;12(4):2129–2135. doi: 10.3892/etm.2016.3608

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Copyright: © Gao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Multi-modal brain magnetic resonance imaging (MRI). (A and B) T1 hypo-intensity in the splenium of the left corpus callosum. (C and D) T1 hypo-intensity in the body of the left corpus callosum. (E and F) T2 hyper-intensity in the splenium of the left corpus callosum. (G and H) T2 hyper-intensity in body of the left corpus callosum. (I-L) Axial diffusion weighted imaging of MRI. (I and J) Diffusion weighted imaging showed high signal intensity in the splenium of the corpus callosum. (K and L) Diffusion weighted imaging demonstrated high signal intensity in the body of the corpus callosum. All these confirm the infarction of left corpus callosum.