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. 2016 Aug 16;12(4):2625–2631. doi: 10.3892/ol.2016.5014

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Copyright: © Jiang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Dynamic changes in the tumor microenvironment during CC progression. (Aa) H&E staining, (Ab) TAM staining with QDs-525 and (Ac) tumor neo-vessel staining with QD-585, in normal tissue. (Ba) H&E staining, (Bb) TAM staining with QDs-525 and (Bc) tumor neo-vessel staining with QD-585, in CC in situ tissue. (Ca) H&E staining, (Cb) TAM staining with QD-525 and (Cc) tumor neo-vessel staining with QD-585, in well-differentiated CC. (Da) H&E staining, (Db) TAM staining with QD-525 and (Dc) tumor neo-vessel staining with QD-585, in poorly-differentiated CC. Aa-Ac, Ba and Bb, Ca-Cc and Da-Dc: Magnification, ×200; and Bc: Magnfication, ×400. TAMs, tumor-associated macrophages; QD, quantum dot; CC, cervical cancer; H&E, hematoxylin and eosin.