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. 2016 Sep 27;9:454. doi: 10.1186/s13104-016-2256-3

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — a Representative western blots showing cellular levels of Src, pSrc, Akt, pAkt, and β-actin in RT112 and RT112^rGEMCI²⁰ cells after 24 h incubation with dasatinib. b Pixel density of western blots is given in percentage compared to untreated cell line (100 %). One of three independent experiments is shown here. *p ≤ 0.05 vs untreated cell line. c Wound healing migration assay. Effects of dasatinib (100 nM) treatment on RT112 or RT112^rGEMCI²⁰ cells were evaluated. Images of the closure of the scratch were captured at 0, 6, and 24 h. Representative images of scratch wound assays were shown (magnification ×100). d Quantification of wound repair was obtained from six measurements of every treatment from three independent experiments