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. Author manuscript; available in PMC: 2017 Sep 27.
Published in final edited form as: Circulation. 2016 Aug 31;134(13):961–977. doi: 10.1161/CIRCULATIONAHA.116.021618

TABLE 1.

In vivo and isolated cell load

control PMAD
ESPVR (mmHg/μl) 1.0±0.10 0.5±0.1 (p=0.02)
PRSW (mmHg) 58±8 30±6 (p=0.02)
CI (mmHg/μl*s) 93±6 64±6 (p=0.005)
τ (ms) 12±3 45±6 (p=7e-11)
ESLTR (au) 84±11 34±6 (p=0.04)
EDLTR (au) 44±6 28±6 (p=0.02)
Peak PRSWcell (au) 8±2 3.0±1 (p=0.006)

ESPVR: end-systolic pressure volume relationship; PRSW: Preload Recruitable Stroke Work; CI: cardiac input; τ: isovolumic relaxation constant; ESLTR: end-systolic length-tension relationship; EDLTR: end-diastolic length-tension relationship.