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. 2016 Oct 3;4:20. doi: 10.1186/s40170-016-0159-3

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© The Author(s). 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — Wnt signaling influences cancer metabolism through regulation of SLC16A1/MCT-1 expression. a Immunohistochemical staining of human colon tumor samples (β-catenin and MCT-1) shows correlations between nuclear β-catenin and high levels of MCT-1. Middle and bottom rows show higher power images of boxed portions in the row above. b Model of a colorectal tumor with Wnt signaling regulating cancer metabolism through target genes including SLC16A1/MCT-1 expression. Examples of Wnt signaling and metabolic inhibitors currently under clinical development shown in boxed regions