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. 2015 Sep 1;237(4):482–494. doi: 10.1002/path.4594

Figure 2.

PATH-4594-FIG-0002-c

Acp1 −/− mice are resistant to pressure overload‐induced hypertrophy and heart failure. Longitudinal assessment of ventricular septum thickness and posterior wall thickness at systole (A, B), end‐systolic volume and end‐systolic diameter (C, D), ejection fraction (E), and fractional shortening (F) measured by echocardiography over 10 weeks after sham operation or TAC in Acp1+/+ or Acp1−/− mice. *p < 0.05, **p < 0.01, ***p < 0.001, and ****p < 0.001 between Acp1+/+ TAC and Acp1−/− TAC animals. † p < 0.05, †† p < 0.01, ††† p < 0.001, and †††† p < 0.001 between Acp1+/+ TAC and Acp1+/+ sham animals (n = 9, Acp1+/+ sham; n = 8, Acp1+/+ TAC; n = 10, Acp1−/− sham; n = 10, Acp1−/− TAC). Statistical analysis was performed using ANOVA.